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MicroRNA-21-5p dysregulation in exosomes derived from heart failure patients impairs regenerative potential

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Without uridine, the cell compensates for pyrimidine shortage by over-activating ATP-consuming salvage pathways (UCK2, TK1, dCK), which potentially drives mitochondrial biogenesis to generate more ATP for these phosphorylation reactions 63

The initial studies towards exploring the radioprotective activity of DSePA were carried out using in vitro models